SYS · ONLINEPASS · 63.0%
Open Assay
Independent Testing / Est. 2026
BATCH04·26·B
PASS63.0%
N27
PeptidesHealingB7-33

B7-33

/ Modified single-chain relaxin-2 analog; biased RXFP1 receptor agonist
SPECULATIVEN = 0 · TESTING PENDINGLAST REVIEW 2026·04·20
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Research use onlyAny dose figures below describe what specific cited studies used, reported factually. Nothing on this page is guidance for human use.READ FIRST →

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§ A · Identity
Primary sequence— sequence not captured —
MW · CLASS · Modified single-chain relaxin-2 analog; biased RXFP1 receptor agonistCATEGORY · Healing

Academic research-phase molecule. Designed to preserve relaxin's anti-fibrotic effects while avoiding vasodilation. No human trials.

§ B · Mechanism of action

B7-33 is a single-chain modified analog of relaxin-2 developed to bias signaling at the RXFP1 receptor toward the Akt/MAPK anti-fibrotic pathway while minimizing cAMP-mediated vasodilation. The biased-agonism hypothesis is that it could preserve cardiac anti-fibrotic benefits of relaxin without the hypotension that limited parent-relaxin (serelaxin) development.

§ C · Human clinical evidence

No human trials. Preclinical mouse models of myocardial infarction and cardiac fibrosis have reported reduced fibrosis markers with B7-33 compared with full-length relaxin. Academic research-phase only.

§ F · Safety signal

No human safety data. The parent molecule (serelaxin) completed Phase 3 trials in acute heart failure that were negative on primary endpoints, though for distinct reasons.

§ H · Regulatory status

Regulatory status

FDA status:
Not FDA-approved
§ I · Notable gaps and controversies

B7-33 is sold in the research-chemical market despite no human trials and no pharmaceutical development program behind it. The biased-agonism rationale is mechanistically interesting but does not substitute for clinical evidence.